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A
Mild muscle weakness reported by the client
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B
Tall peaked T waves on the ECG monitor — hyperkalemia-induced cardiac changes are immediately life-threatening; ventricular fibrillation can occur without warning
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C
A potassium level of 6.2 mEq/L alone
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D
Moderate thirst
Why this is the answer
HYPERKALEMIA (K+ above 5.0 mEq/L) produces a predictable progression of ECG changes that correlates with potassium level and toxicity: K+ 5.5-6.5 mEq/L: PEAKED T WAVES — tall, narrow, symmetrical T waves; this is the first ECG sign; K+ 6.5-7.5 mEq/L: prolonged PR interval, widened QRS, loss of P waves; K+ above 7.5 mEq/L: sinusoidal wave pattern → VENTRICULAR FIBRILLATION → asystole. THE URGENCY: Peaked T waves indicate the cardiac conduction system is being affected and the patient is at immediate risk of fatal dysrhythmia; this takes priority over subjective symptoms like weakness. NURSING PRIORITIES WITH PEAKED T WAVES: (1) CONTINUOUS CARDIAC MONITORING — apply immediately if not already in place; (2) NOTIFY PROVIDER IMMEDIATELY; (3) PREPARE FOR EMERGENCY TREATMENT: Calcium gluconate IV — cardiac membrane stabiliser; does not lower K+ but protects the heart; fastest-acting intervention; Sodium bicarbonate IV — drives K+ into cells (alkalosis shifts K+ intracellularly); Dextrose 50% + Regular insulin — drives K+ into cells (faster-acting); Kayexalate (sodium polystyrene) — GI potassium binder; removes K+ from body; slower (hours); Dialysis for severe refractory hyperkalemia. CAUSES OF HYPERKALEMIA: Renal failure (most common); ACE inhibitors; potassium-sparing diuretics; excessive supplementation; acidosis; tissue destruction (rhabdomyolysis, haemolysis).
Source: NCLEX-RN Physiological Integrity — Hyperkalemia and Cardiac Effects